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Inflammation of the gingiva as a response to bacterial plaque on adjacent teeth; characterized by erythema, edema, and fibrous enlargement of the gingiva without resorption of the underlying alveolar bone. [gingiva + G. -itis, inflammation]
acute necrotizing ulcerative g. (ANUG) See necrotizing ulcerative g.
atypical g. plasma cell g
chronic desquamative g. a clinical term for a gingival condition of unknown etiology, usually encountered in middle-aged and older women, characterized by erythema, mucosal atrophy, and desquamation, and usually accompanied by a burning sensation and pain; diagnosis is usually made by biopsy and direct immunofluorescence.gingivosis;
diabetic g. g. in which the host response to bacterial plaque is presumably modified by the metabolic alterations encountered in the uncontrolled diabetic patient.
dilantin g. diphenylhydantoin g
diphenylhydantoin g. g. exacerbated by long-term therapy with diphenylhydantoin; the host response to bacterial plaque is characterized by marked hyperplasia of the fibrous connective tissue and, to a lesser degree, of the surface epithelium, resulting in gross enlargement of interdental papillae which may coalesce and obscure the clinical crowns of the teeth.dilantin g;
fusospirochetal g. necrotizing ulcerative g
hormonal g. g. in which the host response to bacterial plaque is presumably exacerbated by hormonal alterations occurring during puberty, pregnancy, oral contraceptive use, or menopause.
hyperplastic g. g. of long-standing duration in which the gingiva becomes enlarged and firm due to proliferation of fibrous connective tissue.
leukemic hyperplastic g. enlarged gingiva due to infiltration of leukemic cells and infection from local factors in the face of diminshed host response.
marginal g. g. in which the clinical alterations are confined to the marginal gingiva and do not involve the attached gingiva.
necrotizing ulcerative g. (NUG) an acute or recurrent g. of young and middle-aged adults characterized clinically by gingival erythema and pain, fetid odor, and necrosis and sloughing of interdental papillae and marginal gingiva which gives rise to a gray pseudomembrane; fever, regional lymphadenopathy, and other systemic manifestations also may be present. A fusiform bacillus and Treponema vincentii can be isolated from the gingival tissues in large numbers and are felt to play a significant but poorly defined role in the pathogenesis.fusospirochetal g., trench mouth, ulceromembranous g., Vincent's disease, Vincent's infection;
plasma cell g. intense hyperemic edema and inflammation of the gingiva resulting from a hypersensitivity reaction. A dense plasma cell infiltrate is seen in the lamina propria.atypical g;
proliferative g. inflammatory changes in the gingiva characterized by proliferation of the gingival components.
suppurative g. g. in which a purulent exudate can be expressed from the gingival surface.
ulceromembranous g. necrotizing ulcerative g
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